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Oct 3 16 9:47 AM

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Sharyl Attkisson, Acute flaccid myelitis, Enterovirus D-68, polio
Sharyl Attkisson – Fact check
http://fullmeasure.news/news/cover-story/enterovirus-12-11-2015
Sun. December 13, 2015My notes and comments. Cloudwalker 
Second::15 until this very day it has not been scientifically proven what has caused children to become paralyzed or die, including any virus.
1:52 The doctors may have said that, but it is not true and those who said it apparently didn't know other doctors were saying the cause was unknown.
3:10 - 3:50 Judicial Watch - silence on the part of the government4:05 “first to die”
5:33 states do not include those inflicted prior to Aug. 2014
6: 50 “ front line researchers are still looking for answers “Proof that ACF has no known cause at this time.
Q. What was the goal, intent, of this report?

 

My goal is and always has been, to treat all my animals humanely, to uphold or improve the image of the beef industry, while at the same time maximizing my profit in a safe manner.

Last Edited By: Sir Loin368 Oct 3 16 9:50 AM. Edited 1 time

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#1 [url]

Oct 3 16 6:01 PM

Guillain-Barré syndrome (GBS) is an acute life-threatening disease of the nervous system that leads to sensory disturbances and acute flaccid paralysis. A group of researchers has now shown for the first time that bacteria, which often cause pneumonia, can trigger the autoimmune disease GBS. Antibodies that not only attack the bacteria but also the outer layer of the body's own nerve cells are a critical step in the pathogenesis of GBS after this respiratory infection.

(patho·gen·e·sis/ (path″ah-jen´ĕ-sis) the development of morbid conditions or of disease; more specifically the cellular events and reactions and other pathologic mechanisms occurring in the development of disease.pathogenet´ic )
And now we have a " BACTERIA " sending the human body into " AUTOIMMUNE " causing " ACUTE FLACCID PAEALYSIS.
https://www.sciencedaily.com/releases/2016/10/161003092441.htm

 

My goal is and always has been, to treat all my animals humanely, to uphold or improve the image of the beef industry, while at the same time maximizing my profit in a safe manner.

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Oct 5 16 12:20 PM

Are all news reporter dumber then a bag of hammers or part of the government cover up to conceal the true cause on AFM?

CDC: “-The patients’ symptoms have been most similar to those caused by certain viruses, including poliovirus, non-polio enteroviruses, adenoviruses, and West Nile virus.'

Reporter: “according to the CDC. The cause is unknown, but it has been linked to viral infections like polio and non-polio enteroviruses, adenoviruses, and the mosquito-borne West Nile virus.”

Here is another reporter that is dumber then a bag of hammers or is part of the cover up of the true cause of AFM..
The CDC did NOT say “ it ( meaning AFM ) has been linked to viral infections” etc. The CDC said “ SYMPTOMS have been most similar to those caused by” etc.
http://www.foxnews.com/health/2016/10/04/cdc-reports-rise-polio-like-illness-that-causes-paralysis.html
When the fact is the symptoms are most like  Polioencephalomalacia!

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Oct 5 16 12:47 PM

Polioencephalomalacia
Merck

Polioencephalomalacia (PEM) is an important neurologic disease of ruminants that is seen worldwide. Cattle, sheep, goats, deer, and camelids are affected. The term PEM denotes a lesion with certain gross and microscopic features that are not specific for a particular etiology or pathogenesis. Historically, PEM has been associated with altered thiamine status, but more recently an association with high sulfur intake has been observed. Other toxic or metabolic diseases (eg, acute lead poisoning, sodium toxicosis/water deprivation) can result in PEM as well.

http:w.merckmanuals.com/vet/nervous_system/polioencephalomalacia/overview_of_polioencephalomalacia.html//ww

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#4 [url]

Oct 5 16 3:00 PM

Polioencephalomalacia
Merck

Polioencephalomalacia (PEM) is an important neurologic disease of ruminants that is seen worldwide. Cattle, sheep, goats, deer, and camelids are affected. The term PEM denotes a lesion with certain gross and microscopic features that are not specific for a particular etiology or pathogenesis. Historically, PEM has been associated with altered thiamine status, but more recently an association with high sulfur intake has been observed. Other toxic or metabolic diseases (eg, acute lead poisoning, sodium toxicosis/water deprivation) can result in PEM as well.

http:w.merckmanuals.com/vet/nervous_system/polioencephalomalacia/overview_of_polioencephalomalacia.html//ww

Red saddle
http://www.caes.uga.edu/publications/pubDetail.cfm?pk_id=7650



Excess Sulfur blocks the absorption of copper.
Excess sulfur can also impair animal performance by reducing the availability of other minerals. For example, hydrogen sulfide in the rumen binds with molybdenum to form thiomolybdates. Thiomolybdates bind with copper in the rumen to form an insoluble complex. Some thiomolybdates are absorbed and impair the metabolism of copper in the body. For example, Gooneratne et al. (1989) reported that certain thiomolybdates cause copper to be bound to blood albumins which renders the copper unavailable for biochemical reactions in the body. Price et al., (1987) showed that tri- and tetrathiomolybdates were the sulfur-molybdenum complexes responsible for reducing copper absorption, while the di- and trithiomolybdates had the greatest effect on copper metabolism in the body.
Sulfur also reduces copper absorption by the formation of insoluble copper sulfide in the rumen, independent of the formation of thiomolybdates. Rumen protozoa degrade sulfur amino acids to sulfide which binds to copper to form an insoluble complex. Smart et al., (1986) reported that decreasing the sulfate concentration of drinking water from 500 to 42 ppm, improved the copper status of cattle. These same researchers reported that the 10 ppm copper recommended by the Beef NRC (1996) was not adequate when cattle drank high-sulfur water, which resulted in a total dietary sulfur intake of 0.35%.
http://www.encognitive.com/node/1144
http://www.encognitive.com/node/1144


http://www.encognitive.com/node/1144

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